Truncated ADR58, derived by shortening full-length ADR58 from 71 bases to 31 bases, maintains comparable functional activity to its full-length counterpart. This compound acts as a potent and selective antagonist of human oncostatin M (OSM), effectively blocking OSMs interaction with the gp130 receptor and inhibiting OSM-mediated signaling. It is utilized in studying diseases related to rheumatoid arthritis [1].
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